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Semax
How it works
Semax switches on the brain's own growth factors, protects nerve cells under low oxygen and stress, and keeps calming enkephalins around longer.
Sources
Read the original sources
- Shadrina et al., 2001: Semax in glial cell cultures (Original abstract; cell culture, not people · checked 2026-10-09)
- Semax, an analog of ACTH(4-10) with cognitive effects, regulates BDNF and trkB expression in the rat hippocampus (Abstract · checked 2026-10-13)
- Comparison of the temporary dynamics of NGF and BDNF gene expression in rat hippocampus, frontal cortex, and retina under Semax action. (2010 · Abstract; full methods not assumed · checked 2026-10-13)
- Semax and Pro-Gly-Pro activate the transcription of neurotrophins and their receptor genes after cerebral ischemia (Abstract · checked 2026-10-13)
- Novel insights into the protective properties of ACTH(4-7)PGP (Semax) at the transcriptome level following cerebral ischaemia-reperfusion in rats (Abstract · checked 2026-10-13)
- Semax and Selank inhibit the enkephalin-degrading enzymes from human serum (Abstract · checked 2026-10-13)
- Antistress action of melanocortin derivatives associated with correction of gene expression patterns in the hippocampus of male rats following acute stress (Abstract · checked 2026-10-13)